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Katelyn Connelly, Ph.D.

@kconnelly92.bsky.social

46 followers 110 following 3 posts

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Katelyn Connelly, Ph.D. @kconnelly92.bsky.social · 3d

Congratulations to my labmates @junzhong.bsky.social and Aidan for their new publication on non-coding somatic mutations in PDAC! I’m honored to have been a part of the work & @amundadottir.bsky.social team.

Struan Grant @struangrant.bsky.social · 5d

Congratulations to the @amundadottir.bsky.social team for their #Gut paper reporting a large-scale multiomic analysis of pancreatic ductal adenocarcinoma. Delighted to contribute along with @adwellspenn.bsky.social. @chopresearch.bsky.social @penngenetics.bsky.social

gut.bmj.com/content/earl...

Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene for pancreatic ductal adenocarcinoma

Background The identification and characterisation of somatic cancer driver mutations in the non-coding genome remains challenging. Objective To broadly characterise non-coding driver mutations for p...

gut.bmj.com

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Katelyn Connelly, Ph.D. @kconnelly92.bsky.social · Jul 24

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Katelyn Connelly, Ph.D. @kconnelly92.bsky.social · May 2

I'm excited to share that our functional characterization of the 1p36.33 pancreatic cancer risk locus, is now live in Nature Communications. @amundadottir.bsky.social

www.nature.com/articles/s41...

Allelic effects on KLHL17 expression underlie a pancreatic cancer genome-wide association signal at chr1p36.33 - Nature Communications

Allele-preferential transcription factor binding can influence pancreatic ductal adenocarcinoma risk loci function. Here, the authors show allele-specific JunB and JunD binding at chr1p36.33 and propo...

www.nature.com

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