Monica, Momoninger Enjoyer 🎄
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battypoison.bsky.social
Monica, Momoninger Enjoyer 🎄
@battypoison.bsky.social
30 years old
she/her

@baradhiblue.bsky.social and @cxsmicxddity.bsky.social are my gfs

18+ followers only
#myart for when I draw

toku+birds+boobs(not birds) mainly

icon by @cxsmicxddity.bsky.social
Pinned
Plz Plz remember I share 🔞 things before following! I can only warn you so many times.

I very much believe in sharing things 8 like so I'll 🔄 anything unless bios/replies tell me not to!

I also like to reply, 2 give encouragement, compliments or trying to help. I hope I'm not annoying.
THREADS ⬇️
Reposted by Monica, Momoninger Enjoyer 🎄
Not to be controversial but I think we should use tools and resources to make people's lives less miserable instead of more
December 27, 2025 at 12:17 AM
Reposted by Monica, Momoninger Enjoyer 🎄
Break time
December 27, 2025 at 7:47 PM
Reposted by Monica, Momoninger Enjoyer 🎄
"that art you shared is fetish art!" I literally cannot express how little I gaf about that
December 27, 2025 at 11:44 PM
Reposted by Monica, Momoninger Enjoyer 🎄
No lie I feel like a ton of people come in with this mentality.
December 28, 2025 at 1:52 PM
Reposted by Monica, Momoninger Enjoyer 🎄
What I hate the most about algorithm addicted ppl is the fact that they're all under the idea that you "make it big" by functionally gambling with your posts and hoping enough of them go viral instead of yknow, actually building a solid foundation by networking by talking to your peers shdhdjwj
Oh my god bruh you only have 300 followers here, of course you’re not getting as much engagement
December 28, 2025 at 4:16 PM
Reposted by Monica, Momoninger Enjoyer 🎄
it probably should be looked into how much animal rights activism (specifically when it's made into a particular personal bugbear) has an alarming tendency to be a pipeline to fascism
December 28, 2025 at 4:37 PM
Reposted by Monica, Momoninger Enjoyer 🎄
Kids These Days dont know how many cartoon characters are just Paul Lynde or Paul Lynde impressions
December 28, 2025 at 3:57 PM
Reposted by Monica, Momoninger Enjoyer 🎄
im woke and i say rouge's tits should be bigger
December 28, 2025 at 6:16 PM
Reposted by Monica, Momoninger Enjoyer 🎄
This is exactly why I love Half-Life, bc for as much as it prides itself on a degree of realism for immersion's sake, it still has shit like "right click to fire the shotgun's Second Barrel" and "wear this backpack to longjump like Mario"
people gotta be less afraid to make video game-ass video games. "look how realistic our animations and mechanics are ooo" fuck you, let me tap the same direction with the control stick over and over to make my guy look like he's sliding around
December 28, 2025 at 8:11 PM
Reposted by Monica, Momoninger Enjoyer 🎄
The approach tested here restores balance without pushing levels too high, an important safety distinction. If confirmed in humans, therapies aimed at NAD+ homeostasis could influence treatment strategies for Alzheimer’s and other age-related brain diseases.
December 28, 2025 at 5:14 PM
Reposted by Monica, Momoninger Enjoyer 🎄
blocking amyloid or tau buildup, therapies could focus on strengthening the brain’s own repair systems. Carefully designed human trials will be needed to see if these results translate beyond animal models. Clinically, the study also raises caution about unsupervised NAD+ supplementation.
December 28, 2025 at 5:13 PM
Reposted by Monica, Momoninger Enjoyer 🎄
If brain energy balance can be restored, recovery may be possible, even after symptoms appear. For patients and families, this reframes the disease from a permanent loss to a potentially reversible state under the right conditions. For researchers, these findings open new doors. Instead of only
December 28, 2025 at 5:13 PM
Reposted by Monica, Momoninger Enjoyer 🎄
research team identified dozens that shifted in Alzheimer’s and returned to normal with treatment. These proteins are involved in energy production, synaptic health, and cellular cleanup. They may serve as future biomarkers or drug targets. This work suggests Alzheimer’s MAY NOT be a one-way path.
December 28, 2025 at 5:12 PM
Reposted by Monica, Momoninger Enjoyer 🎄
intact. NDAN brains showed preserved NAD+ balance and healthier protein profiles. In contrast, Alzheimer’s brains showed reduced NAD+ and changes in enzymes that either produce or consume it. Those patterns closely matched what was seen in the mouse models. By mapping thousands of proteins, the
December 28, 2025 at 5:12 PM
Reposted by Monica, Momoninger Enjoyer 🎄
biomarker, returned to normal, strengthening the case for true disease reversal. Researchers ALSO studied brain tissue from PEOPLE with Alzheimer’s and from individuals known as nondemented with Alzheimer’s neuropathology, or NDAN. These individuals had heavy plaque buildup but remained cognitively
December 28, 2025 at 5:11 PM
Reposted by Monica, Momoninger Enjoyer 🎄
brain tissue. Cells that support small blood vessels regained coverage. Markers of inflammation and DNA damage dropped. Synaptic function, measured by electrical signaling in the hippocampus, recovered even when treatment began late. Blood levels of phosphorylated tau-217, a clinical Alzheimer’s
December 28, 2025 at 5:10 PM
Reposted by Monica, Momoninger Enjoyer 🎄
This suggests the brain regained its ability to clear toxic proteins. Tau phosphorylation, a damaging chemical change, also declined. The blood-brain barrier, which normally shields the brain from harmful substances, showed clear repair. Structural gaps closed. Leaky blood proteins stopped entering
December 28, 2025 at 5:09 PM
Reposted by Monica, Momoninger Enjoyer 🎄
normalized after treatment. Restoring the brain's energy balance successfully achieved pathological and functional recovery in both lines of mice with advanced Alzheimer's. The recovery went beyond behavior. Amyloid plaques were reduced, even though amyloid production itself did not change.
December 28, 2025 at 5:09 PM
Reposted by Monica, Momoninger Enjoyer 🎄
Brain structure improved. Memory and learning returned to near normal levels. In maze tests that measure spatial memory, untreated mice struggled. Treated mice performed like healthy controls. In object recognition tests, which measure recall, the same pattern appeared. Even mood-related behaviors
December 28, 2025 at 5:09 PM
Reposted by Monica, Momoninger Enjoyer 🎄
blood-brain barrier breakdown, nerve damage, and behavioral changes similar to those seen in people. When P7C3-A20 was given before symptoms began, it prevented disease features. More striking was what happened when treatment started later. Mice with advanced disease REGAINED normal NAD+ balance.
December 28, 2025 at 5:08 PM
Reposted by Monica, Momoninger Enjoyer 🎄
above safe levels, unlike some over-the-counter supplements. To model Alzheimer’s, researchers used two types of mice. One carried human mutations linked to amyloid buildup. The other carried a human mutation in tau, another hallmark of the disease. Both models developed memory loss, inflammation,
December 28, 2025 at 5:07 PM
Reposted by Monica, Momoninger Enjoyer 🎄
worsened as disease progressed. The team decided to focus on restoring NAD+ homeostasis (meaning healthy balance rather than excessive levels). They used a compound called P7C3-A20, developed in their lab, which helps cells maintain normal NAD+ under stress. Importantly, it does not push NAD+
December 28, 2025 at 5:07 PM
Reposted by Monica, Momoninger Enjoyer 🎄
In Alzheimer’s, the drop is sharper and more damaging. By examining human Alzheimer’s brains AND engineered mouse models, researchers observed that NAD+ balance was SEVERELY disrupted. Cells could no longer meet energy demands or defend themselves against stress. This failure appeared early and
December 28, 2025 at 5:06 PM