Nature Aging
@nataging.nature.com
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Nature Aging is a thematic journal from the Nature Portfolio that serves the broad aging research community. Follows & posts ≠ endorsements.
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🗞️Our September issue is live!📷This month, we're featuring work on somatic mutations, clocks, and more. Read it all here: nature.com/nataging/vol...
nataging.nature.com
🗞️Our September issue is live!📷This month, we're featuring work on somatic mutations, clocks, and more. Read it all here: nature.com/nataging/vol...
nataging.nature.com
🎉 We’re thrilled to present our August issue, which includes a Focus on immune aging! 🎉Here, you’ll find a series of reviews and opinion pieces on recent advances in immune aging research nature.com/nataging/vol... 🎨by Alex Whitworth
nataging.nature.com
🗞️Our July issue is live!📷 This month, we're featuring work on intrinsic capacity, senescence, and more. Read it all here: www.nature.com/nataging/vol...
nataging.nature.com
🗞️Our June issue is live!📷 This month, we're featuring work on editing epigenetic age, somatic mutation, senescence, Alzheimer’s biomarkers and much more. Read it all here: nature.com/nataging/vol...
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Join us in Lisbon for the GIMM Festival: #Aging and #Longevity, happening September 4–6! Explore the future of aging with 20+ invited international experts.
Secure your spot on: gimm.pt/gimm-festival/
@nataging.nature.com
nataging.nature.com
Online now! Researchers @ki.se show that the progeria-causing mutation, LMNA 38 c.1824C>T, is a somatic mutation in arteries from patients with chronic kidney disease, clonal propagation of the mutation in the vascular wall results in vascular aging in mice (rdcu.be/eqv2M)
doi.org/10.1038/s435...
Recurrent somatic mutation and progerin expression in early vascular aging of chronic kidney disease
Nature Aging - Progerin, a truncated version of lamin A, causes Hutchinson–Gilford progeria syndrome characterized by premature aging and cardiovascular complications. Here the authors show...
rdcu.be
nataging.nature.com
Online now! Hu et al., show an aging-related navigational decline owed to deterioration of the excitatory PVMVN → DTN pathway in brainstem. Targeted chemo-genetic pathway activation in aged mice rectified the deficit, highlighting a therapeutic avenue for cognitive aging. doi.org/10.1038/s435...
Harnessing a noncanonical vestibular input in the head-direction network to rectify age-related navigational deficits - Nature Aging
Hu et al. show an aging-related navigational decline owing to deterioration of the excitatory PVMVN→DTN pathway in the brainstem. Targeted chemogenetic pathway activation in aged mice rectified the de...
doi.org
nataging.nature.com
Online now! P-selectin has been considered as a biomarker of hematopoietic stem cell (HSC) aging. Here, Yang et al., uncovered a novel functional role of P-selectin engagement in regulating HSC regeneration and driving stem cell aging when perturbed. doi.org/10.1038/s435...
Aberrant engagement of P-selectin drives hematopoietic stem cell aging in mice - Nature Aging
P-selectin has been considered as a biomarker of hematopoietic stem cell (HSC) aging. Here, Yang et al. uncovered a new functional role of P-selectin engagement in regulating HSC regeneration and driv...
doi.org