niels-odum.bsky.social
@niels-odum.bsky.social
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niels-odum.bsky.social
niels-odum.bsky.social @niels-odum.bsky.social · Jun 30
Congratulations Terkild and the team👏💪😊 very important new knowledge on the intricacies of this horrible cancer - this new understanding of coexisting yet specialized cancer subclones is a key discovery for a truly targeted treatment in the future !!!!
Bravo 👏👏👏
terkild.bsky.social
Terkild Brink Buus @terkild.bsky.social · Jun 19
❗How can a cancer exploit its environment and still resist treatment?
✅The answer: co-existing malignant subclones.

Let me walk you through our latest study investigating how divergent evolution drives adaptability, aggressiveness, and drug resistance of T cell cancer. 1/🧵

doi.org/10.1158/2159...
Schematic figure showing divergent evolution of malignant subclones and how they migrate to different tissue compartments (blood vs skin), respond differently to extrinsic stimuli such as Staphylococcus aureus enterotoxins, and how subclones differ in their intrinsic resistance to anti-cancer drugs.
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niels-odum.bsky.social
niels-odum.bsky.social @niels-odum.bsky.social · Jun 20
Congratulations with this impressive and important work 👍👏💪
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niels-odum.bsky.social
niels-odum.bsky.social @niels-odum.bsky.social · Nov 28
Cutaneous T cell lymphoma - yet surprisingly little is known on how malignant T cells communicate with keratinocytes - Ziao Zeng has now shown how presentation of staphylococcal toxins by keratinocytes may fuel the cancer - now published in December issue of JID www.sciencedirect.com/journal/jour...
Journal of Investigative Dermatology | Vol 144, Issue 12, Pages A1-A8, 2609-2836 (December 2024) | ScienceDirect.com by ElsevierScienceDirect
Read the latest articles of Journal of Investigative Dermatology at ScienceDirect.com, Elsevier’s leading platform of peer-reviewed scholarly literature
www.sciencedirect.com
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