Artur Vetkas
@ca1ca2ca3ca4.bsky.social
Optimal SCS enhances PAG and midbrain activity while reducing signals in emotional/cognitive hubs like the ACC and insula. On a network level it strengthens descending pain inhibition and modulates affective/cognitive pain pathways for effective analgesia. (3/4)
January 18, 2025 at 5:49 PM
Optimal SCS enhances PAG and midbrain activity while reducing signals in emotional/cognitive hubs like the ACC and insula. On a network level it strengthens descending pain inhibition and modulates affective/cognitive pain pathways for effective analgesia. (3/4)
We saw that paresthesia-free or low-frequency (40 Hz) stimulation augments prefrontal engagement, while high-frequency (1200 Hz) modulates sensorimotor cortices. (2/4)
January 18, 2025 at 5:49 PM
We saw that paresthesia-free or low-frequency (40 Hz) stimulation augments prefrontal engagement, while high-frequency (1200 Hz) modulates sensorimotor cortices. (2/4)
Chronic pain arises from interactions among sensory, cognitive, and emotional circuits. Our recent fMRI investigation published in Brain Stimulation of SCS reveals how neuromodulation may help restore equilibrium in these central pain pathways. (1/4) t.co/VFH7dfHb0s
January 18, 2025 at 5:48 PM
Chronic pain arises from interactions among sensory, cognitive, and emotional circuits. Our recent fMRI investigation published in Brain Stimulation of SCS reveals how neuromodulation may help restore equilibrium in these central pain pathways. (1/4) t.co/VFH7dfHb0s