Christin Herrmann
@christinviral.bsky.social
440 followers 460 following 11 posts
Scientist with Victor Torres in the Department of Host Microbe Interactions at St. Jude. Postdoc in the Cadwell Lab (NYU and UPenn) and grad school in the Weitzman Lab (UPenn).
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Reposted by Christin Herrmann
erinrgreen.bsky.social
Job alert ‼️ UChicago Micro is hiring! Open to tenured/tenure track faculty at all levels in any area of microbiology. Come join our amazing and growing department. apply.interfolio.com/174404
Reposted by Christin Herrmann
immunuelle.bsky.social
I'm thrilled to announce I'll be joining the Department of Molecular Genetics & Microbiology at the University of Florida in January 2026! My lab will study RNA virus-host interactions with a particular focus on the spatial regulation of viral replication and innate immune activation. genoyerlab.com
Genoyer Lab
genoyerlab.com
christinviral.bsky.social
Congrats! That's awesome news.
Reposted by Christin Herrmann
joeyzacks.bsky.social
Excited to share this collaborative study with @ritatamayo.bsky.social showing that E. faecalis influences C. diff morphology through the phase variable CmrRST system! This work was co-led by the amazing team of @ashleyweiss.bsky.social and Jilarie Santos-Santiago!
biorxiv-microbiol.bsky.social
Enterococcus faecalis modulates phase variation in Clostridioides difficile https://www.biorxiv.org/content/10.1101/2025.07.24.666506v1
christinviral.bsky.social
Thank you to all of my amazing co-authors in the @cadwelllab.bsky.social and Koralov lab!
christinviral.bsky.social
In addition, we also see these inflammatory responses after cage change in mock mice. This highlights that we should be aware of the side effects common mouse husbandry practices can introduce into experiments.
christinviral.bsky.social
This initially represses immune responses and allows virus expansion. Once stress is resolved, CD8 T cells in the gut become activated and induce inflammatory responses in the epithelium leading to virus clearance. Therefore, manipulating the environment we can shorten the duration of an infection.
christinviral.bsky.social
We noticed variability in the length of infection for one of our enteric model viruses, murine Astrovirus, and discovered that the virus was cleared because of the stress following cage changes. The new environment leads to production of stress hormones such a glucocorticoids.
Reposted by Christin Herrmann
joeyzacks.bsky.social
Incredibly honored to be selected as a @bwfund.bsky.social Investigator in the Pathogenesis of Infectious Disease. Thank you to @bwfund.bsky.social for supporting our work - I am thrilled to join this amazing community! And special thank you to my AMAZING team (past & present) for getting us here!
Reposted by Christin Herrmann
cadwelllab.bsky.social
Congratulations Xiaomin Yao and Eugene Rudensky for publishing this story @cp-immunity.bsky.social! Big thank you to NIH for making this work possible. Here’s a brief summary of this 🤯 finding about a gene variant that YOU 🫵 most likely have 1/n
kwnsfk27.r.eu-west-1.awstrack.me
Reposted by Christin Herrmann
reyesruiz.bsky.social
1/3 It’s official 🙌🏽
The Reyes Ruiz Lab is opening in 2026 at the University of Wisconsin, Madison in the Department of Pathobiological Sciences. My lab will focus on host antibacterial effectors and adaptation of bacteria to host-imposed stress during S. aureus infections.
Reposted by Christin Herrmann
carman-mc-li.bsky.social
Our lab has opened at the Cancer Biology Department and the Basser Center for BRCA @upenn.bsky.social! We study new mechanisms of hereditary cancers and their implications for sporadic cancers, building on my training with @labjacks.bsky.social and Joan Brugge.

Join us! tinyurl.com/CarmanLiLabH...
Carman Li Lab
www.CarmanLiLab.org
Reposted by Christin Herrmann
biorxiv-microbiol.bsky.social
Gastrointestinal colonization as a source of Staphylococcus aureus in atopic dermatitis https://www.biorxiv.org/content/10.1101/2025.04.17.648849v1
Reposted by Christin Herrmann
scottehensley.bsky.social
After 8 months of peer review, our manuscript was published today @nature.com showing that bovine H5N1 viruses bind poorly to human-type sialic acid receptors. We now know that the virus is only 1 HA substitution away from efficiently binding human receptors.

www.nature.com/articles/s41...
Bovine H5N1 binds poorly to human-type sialic acid receptors - Nature
Nature - Bovine H5N1 binds poorly to human-type sialic acid receptors
www.nature.com